Powdery scab is an illness of potato tubers. It is brought about by the cercozoan Spongospora subterranea f. sp. subterranea and is broad in potato developing nations. Indications of Powdery scab remember little sores for the beginning phases of the sickness, advancing to raised pustules containing a Powdery mass. These can ultimately burst inside the tuber periderm. The Powdery pustules contain resting spores that discharge anisokont zoospores (abiogenetic spore with two inconsistent length flagella) to contaminate the root hairs of potatoes or tomatoes. Powdery scab is a corrective deformity on tubers, which can bring about the dismissal of these potatoes. Potatoes which have been tainted can be stripped to eliminate the contaminated skin and the leftover within the potato can be cooked and eaten. By and large, not a ton is thought about the existence pattern of Spongospora subterranea f. sp subterranea (SSS). The vast majority of the presently proposed life cycle depends on that of Plasmodiophora brassicae, a firmly related and better-considered protozoan. It has been proposed, because of this likeness, that there are two particular stages that SSS can exist as; the agamic and sexual stages. Agamic Stage: A zoospore taints root tissue and turns into a uninucleate plasmodium. This plasmodium goes through mitotic atomic division and transforms into a multinucleate plasmodium. Then, at that point, the multinucleate plasmodium structures zoosporangium, which ultimately discharge more zoospores. This interaction can happen generally rapidly and can go about as a significant wellspring of auxiliary inoculum inside a field.

Sexual Stage: This stage follows a comparative example to the agamic stage, yet with a couple of exemptions. It is estimated that two zoospores intertwine to frame a dikaryotic zoospore and afterward taint the roots. When the contamination happens, the dikaryotic zoospore forms into a binucleate plasmodium. Like the abiogenetic stage, this plasmodium will likewise reproduce its core to make a multinucleate plasmodium. The second fundamental diverse between stages happens here. The sets of cores will meld by karyogamy, and the plasmodium will rapidly partition into various resting spores inside a sporosori. These resting spores have three-layered dividers and are very impervious to the climate, permitting them to persevere in the dirt for more than 10 years. As an update, a large portion of the existence cycle is as yet hazy. Be that as it may, the presence of zoospores, plasmodia, zoosporangia, and resting spores have been seen in the field and lab. The ploidy levels and karyogamy occasions are just guessed and still can't seem to be demonstrated.

Spongospora subterranea pathogenesis is best in cool, soggy conditions, like northern Britain, the Columbia Basin of south-focal Washington, and north-focal Oregon. The ecological condition is especially basic during the arrival of infective specialists (zoospores) into the dirt climate. Upon discharge from resting spores, zoospores expect dampness to swim towards the host tuber or roots. One examination, discovered Powdery scab was altogether more normal on plants filled in steady sogginess contrasted with plants developed with fluctuating dampness levels. In this equivalent examination she closed illness hazard was connected more to the climate, or dampness level, than the degree of inoculum present. Inoculum might be available yet not ready to scatter because of natural conditions, and accordingly doesn't arrive at have tissue to taint. Other natural factors that influence Spongospora subterranea contamination are straightforwardly identified with agronomic practices. Expanded utilization of manures containing nitrate or ammonium nitrogen increment the occurrence and seriousness of Powdery scab. It is believed that the treatment expands root development, and accordingly gives more tissue to contamination and sickness cycling to happen. Likewise, diminished cellulose inside the cell dividers brought about by overabundance nitrogen may expand vulnerability of host to disease. It is evident that the climate can straightforwardly influence both the host powerlessness and the dispersal of the microbe at last establishing the rhythm for the infection cycle.

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